I suspect most of us try to avoid placing stents in the central venous system , but unfortunately sometimes those situations do arise. Factors such as covered vs bare metal (horrible instent restenosis but at least permit flow from neighboring veins), capacity of radial force to help hold a lesion open, stents that lengthen/forshorten, etc.
Tthoughts on our current available products/technologies to tackle these tough cases?
Posted Monday, October 31, 2016
I am glad you opened this discussion. The management of central venous stenosis is a pivotal issue second in importance only to the issue of prevention of central venous stenosis, namely by avoiding or limiting the use of central venous catheters in the first place (NKF-DOQI, '97).
We have found the use of stent grafts (specifically the Viabahn stent) in the central veins to be consistent with long term patency, and presented our results in poster and podium form at CIDA and VASA meetings in the past. A 90% five year secondary patency of stented central veins in 142 patients was documented. We now have over 200 patients in our little registry. I shake my head at meetings where participants have qualms about central venous stenting, and are apparently unaware that a large experience has already been presented several times. I must be a very poor speaker, or have lost my credibility somehow.
Bare wire stents are prone to fracturing, tissue ingrowth, and failure. We do not use them except the very biggest sizes in the innominant or SVC. If they are all we have at the moment, we resign ourselves to re-lining the barewire stent with a stent-graft. I reject the argument that barewire stents prevent "jailing out" side branches - if the channel goes down, everything is jailed out anyway.
Wall Stents and Wall Grafts have variable lengths when deployed and expanded - we don't use them any more. The Bard Fluency/Flair and Gore Viabahn stents do not have that problem (they have other problems). We use mostly Viabahn stents - I have placed several thousand since 2000.
I prefer to stent open the central veins rather than putting in a HeRO graft/catheter. Every time the patient moves their arm, the distance between the fixed end in the upper arm or shoulder and the free end in the atrium changes. The net effect is that a long stiff catheter component is bobbing up and down in the atrium. On occasion, the free end may get caught in the coronary sinus on the posterior wall of the atrium - and then your risk of death multiplies. The HeRO is a glitzy and cheap but dangerous solution that is most often more effectively and safely provided by a stent.
I could talk all night, but the men have come to lock me in my room for the evening again. Maybe tomorrow we can talk about the V-Wing, another dubious "advance".